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Hugo Barra, Director of Google Product Management, holds up the new Google Nexus7 tablet at the Google I/O conference in San Francisco, Wednesday, June 27, 2012. (AP Photo/Paul Sakuma)

Hugo Barra, Director of Google Product Management, holds up the new Google Nexus7 tablet at the Google I/O conference in San Francisco, Wednesday, June 27, 2012. (AP Photo/Paul Sakuma)

Hugo Barra, Director of Google Product Management, holds up the new Google Nexus7 tablet at the Google I/O conference in San Francisco, Wednesday, June 27, 2012. (AP Photo/Paul Sakuma)

Hugo Barra, Director of Google Product Management, holds up the new Google Nexus7 that will sell for $199, at the Google I/O conference in San Francisco, Wednesday, June 27, 2012. The tablet will have a screen that measures 7 inches diagonally, smaller than the nearly 10 inches on Apple Inc.?s popular iPad. That means it?s more likely to challenge Amazon.com Inc.?s Kindle Fire, which is also 7 inches. (AP Photo/Paul Sakuma)

Hugo Barra, Director of Google Product Management holds up the new Google Nexus7 selling at $199, at the Google I/O conference in San Francisco, Wednesday, June 27, 2012. The tablet will have a screen that measures 7 inches diagonally, smaller than the nearly 10 inches on Apple Inc.?s popular iPad. That means it?s more likely to challenge Amazon.com Inc.?s Kindle Fire, which is also 7 inches. (AP Photo/Paul Sakuma)

Hugo Barra, Director of Google Product Management, holds up the new Google Nexus7 tablet at the Google I/O conference in San Francisco, Wednesday, June 27, 2012. (AP Photo/Paul Sakuma)

SAN FRANCISCO (AP) ? Google unveiled its attempt to catch up to Apple and Amazon in the growing market for tablet computers. It also provided a titillating glimpse at its vision of a digital future through the mind-boggling lens of Internet-connected glasses.

The debut of a long-anticipated tablet computer bearing Google’s brand and equipped with its latest operating software kicked off an annual conference for about 6,000 computer programmers.

Although the tablet drew plenty of applause, the biggest crowd-pleasing moment came near the end of a nearly two-hour performance Wednesday when Google conjured a scene worthy of a “Mission Impossible” movie.

After Google co-founder Sergey Brin trotted on stage during the middle of a presentation, he cut away to a live video feed showing a group of skydivers the company hired to jump out of a blimp hovering about 7,000 feet above downtown San Francisco.

The skydivers were wearing a pair of Internet-connected glasses. That allowed the audience inside to see what the skydivers were seeing as they parachuted on to the roof of the building where the conference was being held.

The glasses are still a work in progress, although Google gave U.S. programmers attending the conference a chance to order a $1,500 prototype that they can start experimenting with early next year.

The Google-branded tablet, called the Nexus 7, will start shipping next month in a direct challenge to Amazon.com Inc.’s Kindle Fire. The tablet also could appeal to consumers looking for a less expensive, less sophisticated alternative to Apple Inc.’s iPad.

The Nexus 7 is designed specifically for Google Play, an online store that sells movies, music, books, apps and other content. That mirrors Amazon’s strategy with the Kindle Fire, although Amazon’s strength in online retailing has seeded its store with a more extensive selection than Google Play. Amazon declined comment.

The size and price of the Nexus 7 also matches the Kindle Fire. Both have 7-inch screens and sell for $199. The Nexus 7 is slightly lighter at about 0.75 pound, compared with the Kindle Fire’s 0.9 pound.

By contrast, the iPad’s screen measures nearly 10 inches diagonally and weighs 1.44 pounds. Apple sells its latest models for $499 and up, though an older version is available for $399.

Customers can start ordering the Nexus 7 through Google on Wednesday, initially in the U.S., Canada and Australia. The device won’t ship until mid-July.

Google’s announcement that it’s putting its brand on a tablet comes a week after Microsoft Corp. did the same thing. Both moves risk alienating Google’s and Microsoft’s hardware partners. Those companies, in turn, could be less inclined to work closely with Google and Microsoft.

The Nexus 7′s price looks like a relative bargain, given that it boasts more features than the Kindle, including a front-facing camera with 1.2 megapixels. The Kindle is believed to be a roughly break-even product for Amazon at $199. Samsung Electronics Co. sells a tablet similar to Google’s for $250.

Andrew Rassweiler, an analyst with IHS iSuppli, said he suspects Google will be selling the Nexus 7 at a loss.

Google has previously put its own brand on a flagship line of “Nexus” smartphones. But that market is more mature than the tablet market, and there was less risk of Google alienating partners, particularly because it didn’t price the phones lower than the norm.

Much like the Nexus phones, the Nexus 7 tablet will be a showcase for a new version of Google’s Android operating system ? this one called Jelly Bean.

Although the tablet carries the Google brand, the machine will be made by AsusTek Computer Inc. Google recently expanded into the device-making business with its $12.5 billion purchase of Motorola Mobility, but the company has stressed that it intends to continue to rely on Asus and other manufacturers that have embraced Android.

Jeff Orr, an analyst with ABI Research, said Microsoft’s announcement of its Surface tablet last week and Google’s Nexus 7 add up to a “troubling” situation for tablet makers such as Samsung Electronics Co., which makes the Galaxy line.

When a software-supplying partner turns around and puts out its own hardware product, “is that a partner or an enemy?” Orr asked.

Orr also questioned whether Google’s strategy of pricing the tablet low is really going to win it any fans in the long term. Apple, he noted, dominates the tablet market with a product that’s expensive but works well.

There are already other Android-powered tablets on the market, but none have proven nearly as popular as the iPad or Kindle Fire. That has raised worries at Google as more people rely on tablets to surf the Internet.

For Google, advertising dollars are at stake. If Apple retains its dominance and other players such as Amazon and Microsoft gobble up the rest of the sales, they could set up their operating systems in ways that de-emphasize Google’s Internet search engine and other services. Apple develops its own system, while Amazon modifies Android for use in Kindles. Microsoft’s will run on a new version of Windows.

Apple already has announced that the next version of the iPad operating system will abandon Google’s digital maps as the built-in navigation system. That shift could cause neighborhood merchants to spend less money advertising on Google.

The iPad currently has about 68 percent of the tablet market, according to Forrester Research. The research firm expects the increasing competition to whittle the iPad’s share to 53 percent in 2016, with Android-powered tablets at about 30 percent and Windows tablets accounting for the remainder. Forrester predicted 760 million tablets will be in use by 2016.

“Google’s user base for music, books, and movies is not nearly as strong as Apple or Amazon, so it will take time to build a strong customer base” for the Nexus 7, predicted Forrester analyst Frank Gillett.

He also said the Nexus 7 and other Android tablets are at a disadvantage because computer programmers still haven’t built as many compelling applications for that platform as they have for the iPad.

In another expansion into consumer electronics, Google also announced a home entertainment device called Nexus Q that is similar to Apple TV, a small box that can stream music and movies over Internet connections. The Nexus Q sends content from your personal collection or YouTube to your existing TV and speaker systems. You control it through a separate Android phone or tablet.

The Nexus Q, which Google is calling the world’s first “social streaming device,” will be available in July in the U.S. initially and sell for $299. Apple TV currently sells for just $99. Other Internet-streaming devices, such as the Roku, that connect to TVs sell for even less than that.

The Internet-connected glasses that Google demonstrated won’t be cheap either, although Brin told reporters Wednesday that the company plans to charge consumers significantly less than the $1,500 that developers had to pay for prototypes. The glasses, which Google is calling Project Glass, probably won’t be released to the mass market until early 2014, Brin said.

“It’s not a commodity product,” Brin said. “We view this as a premium sort of thing.”

Google began developing the Internet-connected glasses in a secret research lab more than two years ago and announced what it was working on in April. Wednesday’s presentation marked the first extended demonstration of how the glasses might work.

Also on Wednesday, Google unveiled a new search tool to help you get the right information at the right time on your mobile device. Called Google Now, the tool will be part of Jelly Bean, which will be available in mid-July. Some devices, including the Galaxy Nexus, will get the upgrade automatically over the air.

With Google Now, if you say “traffic,” for example, it will look at your usual commute to work and show you alternative routes if there’s a lot of traffic. It will tell you the scores of your favorite sports teams automatically, and it will keep you up to date on flight statuses if you are flying somewhere.

The feature bears resemblance to the Siri virtual assistant on Apple’s iPhone.

Jelly Bean will also come with the ability to share photos by tapping two phones together.

___

Peter Svensson contributed from New York. AP Technology Writer Barbara Ortutay in New York contributed to this story.

Associated Press

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28 June 2012 Last updated at 14:12 ET By Pallab Ghosh Science correspondent, BBC News

The oldest known samples of pottery have been unearthed in southern China.

The US archaeologists involved have determined that fragments from a large bowl found in Xianrendong Cave, Jiangxi Province, are 20,000 years old.

The discovery, published in the journal Science, is the latest in recent years that have pushed back the invention of pottery by 10,000 years.

It is thought that the bowl was a cauldron to cook food, or possibly to brew alcohol.

Until recently, the majority view was that pottery bowls and drink receptacles were invented after the emergence of agriculture, when people began to stay in one place for long periods.

Part of the reasoning was that pottery items are large and breakable, and so not a useful technology for hunter-gatherer societies that moved from place to place in search of food.

Continue reading the main story

?Start Quote

People were gathering together in larger groups and you needed social activities to mitigate against increased tensions. Maybe the potteries were used to brew alcohol?

End Quote Prof Gideon Shelach Hebrew University

But in the past 10 years, researchers have found instances of pottery pre-dating agriculture.

One possible reason for the invention of pottery is that 20,000 years ago the Earth was the coldest it had been for a million years.

According to the lead researcher, Prof Ofer Bar-Yosef of Harvard University, pottery cauldrons would have enabled people to extract more nutrition from their food by cooking it.

“Hunter-gatherers were under pressure to get enough food,” he told BBC News.

“If the invention is a good one, it spreads pretty fast. And it seems that in that part of southern China, pottery spread among hunter-gatherers in a large area.”

Prof Gideon Shelach of the Hebrew University in Jerusalem speculates that there may also have been a social driver for the invention of pottery.

“People were gathering together in larger groups and you needed social activities to mitigate against increased tensions,” he told BBC News.

“Maybe those potteries were used to brew alcohol.

“It used to be thought that the beginning of pottery was associated with agriculture and sedentary lifestyle,” he added.

“Yet here we find it 8,000 years or more before this transition. This is a very puzzling situation.”

The archaeological team estimates from fragments of the bowl that it was 20cm high and 15-25cm in diameter.

Prof Bar-Yosef is keen to discover what these ancient people were cooking 20,000 years ago. He believes that whatever it was it was either steamed or boiled in the bowl.

“It’s probably not the first wok,” he said. “Cooking with oil began later.

“We think it was used for cooking with water, so it is more like a caldron”.

Follow Pallab on Twitter

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Here she is, folks. The Google I/O 2012 T-shirt, done up in the same machine-like tools we've come to know in love from Google in the past six months or so.

And after the break, a little snippet of code. Anyone want to tell us what it does?

Oh, and by the way, we're giving away a couple of these shirts. If you want in on the action, check out the video in this post.

read more

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We're pretty sure that Google TV is going to feature quite prominently at Google I/O — just in case you forgot, we'll be there all week! — and to go along with that, the Google TV website got all refreshed. It falls into Google's general way of designing their web pages these days. It looks clean, simple, and fresh. It also gives a good breakdown of the devices available, both "buddy box" and integrated devices. There's also a decent guide to what Google TV actually is, and the applications that you can enjoy. 

At the end of the day, it's just a website. But, for us, now lucky Brits — and indeed several of our international cousins — at least, Google TV is new and exciting. Being able to watch Eastenders and browse the web at the same time? Sounds good. 

Oh, care for a dose of speculation? Well, when you're taking a look around the devices section of the site, a little feller in an I/O 12 t-shirt pops up behind a newly announced, Vizio Google TV box. Probably means we'll be seeing quite a bit of this little guy over the next few days as well. 

Source: Google TV

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. . . go get yourself some new talking points.

Seriously, the old “Oh, well, things have changed in the past, so what’s the worry?” canard?

The processes you describe took place over millions of years.

We’re talking relatively drastic changes, over the course of decades, on a highly developed area of an increasingly crowded and interdependent planet.

If a drunk driver speeding through a red light ran over your dog or your kid, would you accept the driver saying, “Look, people die in accidents all the time. In seventy years, a trivial fraction of the age of the Earth, your kid would likely be dead anyway. Calm down and accept change as a normal part of life. And anyway, can you really prove it was my car that killed your kid? Maybe you wiped his blood on my bumper so you could sue me, and infringe on my right to drink and drive!”

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Public release date: 24-Jun-2012
[ | E-mail | Share ]

Contact: Mary Rice
mary.rice@riceconseil.eu
European Society of Human Genetics

Nuremberg, Germany: The use of genome-wide array analysis[1] in parents whose children are suspected of having a genetic disease shows that the parents frequently also have previously undetected genetic abnormalities, a researcher from The Netherlands told the annual conference of the European Society of Human Genetics today (Sunday). Being aware of this is important to parents because it means that their risk of having another affected child is significantly increased.

Dr. Nicole de Leeuw, a clinical laboratory geneticist in the Department of Human Genetics of the Radboud University Nijmegen Medical Centre in Nijmegen, and colleagues performed genome-wide SNP[2] array analysis in 6,500 patients and 1,874 parents. The patients had intellectual disability and/or congenital abnormalities, and the parents of those in whom an aberration was detected were tested in a similar way to determine whether they had the same aberration as their child. Mosaic aberrations, where both genetically normal and abnormal cells are present in an individual, were not only found in one in every 300 patients, but in one in every 270 parents as well. “These abnormalities occurred more frequently than we had expected”, said Dr. de Leeuw. “Armed with this knowledge, we can try to understand not only why, but also how genetic disease arises in individuals, and this can help us to provide better genetic counselling.”

Analysis of patients’ genomes showed 6.5% de novo (spontaneously arising) genomic imbalances, 9.1% of rare, inherited imbalances, and 0.8% of X-linked abnormalities. Moreover, with the additional data from their SNP array test results, the researchers were able to subsequently find pathogenic mutations in recessive disease genes, uniparental disomies (where a single chromosome is doubled leading to two genetically identical ones), and mosaic aneuploidies (an extra or missing chromosome in some of the cells of the body) in about 30 patients.

“In at least seven families, these findings meant that what we had thought of as a spontaneously arising, non-inherited genetic abnormality in a child was in fact already present in some form in the parent”, said Dr. de Leeuw. “Furthermore, when we tested in different cell lines for example, DNA from blood and that from a mouth swab we often found that results varied. This is because mosaic aberrations can occur in cells in some organs and not in others, and underlines the importance of not just relying on one type of cell line for this kind of genetic diagnosis.”

In two cases these tissue-dependent differences changed over time, and the researchers believe that this was due to an attempt by the body to correct and rescue the situation. “Such rescue attempts are best known in cases of trisomy, where there are three chromosomes instead of two in a cell, or monosomy, where there is only one. In both these cases, the body may try to correct the situation by respectively deleting or adding (doubling) a chromosome. Such rescue mechanisms may be more common than we expected, and by using genome-wide SNP array analysis it will help us to reveal them. For some patients, it would be particularly interesting if we could test multiple samples of these patients over time”, said Dr. de Leeuw.

The majority of genetic diseases are not treatable, but in some cases a special diet may reduce the severity of the symptoms ,for example, in phenylketonuria (PKU) or in coeliac disease, in others the same can be obtained by periodic examination of certain organs (for example in Down syndrome or Marfan syndrome). Sometimes hormone treatment will be of benefit to the patient, for example growth hormone treatment in Turner syndrome. For most patients with a genetic disorder, there is no cure, but knowing the genetic cause of their disease may help and improve the care for these patients through knowledge about other patients with the same disease. And if the family is at risk of a genetic disease, couples considering having children can be better informed as to their options, the researchers say.

“By using genome-wide array analysis to look for imbalances in the human genome, we will uncover more and more accurate findings in patients. This will not only increase our knowledge of genetic disorders and the human genome in general, but if we can also collect the clinical features of these patients in a structured and uniform way, the information will become increasingly valuable. Fortunately, this is becoming easier due to advances in tools and software applications, and many professionals in the academic and commercial world have agreed to collaborate in order to substantially increase the genotype/phenotype collection and make these anonymised data publicly available to medical professionals in order to improve patient care worldwide”, Dr. de Leeuw concluded.

###

Abstract no: C04.5, Session: Sunday, June 24, 1:15 pm

[1] A DNA microarray (or gene chip) is a collection of microscopic DNA spots attached to a solid surface, used to measure the relative quantity of large numbers of genes simultaneously in multiple regions of a genome, thereby determining the DNA copy number.

[2] A SNP (pronounced ‘snip’), or single nucleotide polymorphism, is a relatively common DNA sequence variation, a change in a single nucleotide A, T, C or G in the genome, without clinical consequences. Each person has about 3 million SNPs in his or her genome, a unique genetic signature. In a SNP microarray, the probes (small pieces of DNA) bind only to the complementary piece of DNA being analysed if it is an exact match, making it possible to determine the genotype (SNP composition) of a DNA sample in addition to the DNA copy number.

---

[ | E-mail | Share ]

?

AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.

Public release date: 24-Jun-2012
[ | E-mail | Share ]

Contact: Mary Rice
mary.rice@riceconseil.eu
European Society of Human Genetics

Nuremberg, Germany: The use of genome-wide array analysis[1] in parents whose children are suspected of having a genetic disease shows that the parents frequently also have previously undetected genetic abnormalities, a researcher from The Netherlands told the annual conference of the European Society of Human Genetics today (Sunday). Being aware of this is important to parents because it means that their risk of having another affected child is significantly increased.

Dr. Nicole de Leeuw, a clinical laboratory geneticist in the Department of Human Genetics of the Radboud University Nijmegen Medical Centre in Nijmegen, and colleagues performed genome-wide SNP[2] array analysis in 6,500 patients and 1,874 parents. The patients had intellectual disability and/or congenital abnormalities, and the parents of those in whom an aberration was detected were tested in a similar way to determine whether they had the same aberration as their child. Mosaic aberrations, where both genetically normal and abnormal cells are present in an individual, were not only found in one in every 300 patients, but in one in every 270 parents as well. “These abnormalities occurred more frequently than we had expected”, said Dr. de Leeuw. “Armed with this knowledge, we can try to understand not only why, but also how genetic disease arises in individuals, and this can help us to provide better genetic counselling.”

Analysis of patients’ genomes showed 6.5% de novo (spontaneously arising) genomic imbalances, 9.1% of rare, inherited imbalances, and 0.8% of X-linked abnormalities. Moreover, with the additional data from their SNP array test results, the researchers were able to subsequently find pathogenic mutations in recessive disease genes, uniparental disomies (where a single chromosome is doubled leading to two genetically identical ones), and mosaic aneuploidies (an extra or missing chromosome in some of the cells of the body) in about 30 patients.

“In at least seven families, these findings meant that what we had thought of as a spontaneously arising, non-inherited genetic abnormality in a child was in fact already present in some form in the parent”, said Dr. de Leeuw. “Furthermore, when we tested in different cell lines for example, DNA from blood and that from a mouth swab we often found that results varied. This is because mosaic aberrations can occur in cells in some organs and not in others, and underlines the importance of not just relying on one type of cell line for this kind of genetic diagnosis.”

In two cases these tissue-dependent differences changed over time, and the researchers believe that this was due to an attempt by the body to correct and rescue the situation. “Such rescue attempts are best known in cases of trisomy, where there are three chromosomes instead of two in a cell, or monosomy, where there is only one. In both these cases, the body may try to correct the situation by respectively deleting or adding (doubling) a chromosome. Such rescue mechanisms may be more common than we expected, and by using genome-wide SNP array analysis it will help us to reveal them. For some patients, it would be particularly interesting if we could test multiple samples of these patients over time”, said Dr. de Leeuw.

The majority of genetic diseases are not treatable, but in some cases a special diet may reduce the severity of the symptoms ,for example, in phenylketonuria (PKU) or in coeliac disease, in others the same can be obtained by periodic examination of certain organs (for example in Down syndrome or Marfan syndrome). Sometimes hormone treatment will be of benefit to the patient, for example growth hormone treatment in Turner syndrome. For most patients with a genetic disorder, there is no cure, but knowing the genetic cause of their disease may help and improve the care for these patients through knowledge about other patients with the same disease. And if the family is at risk of a genetic disease, couples considering having children can be better informed as to their options, the researchers say.

“By using genome-wide array analysis to look for imbalances in the human genome, we will uncover more and more accurate findings in patients. This will not only increase our knowledge of genetic disorders and the human genome in general, but if we can also collect the clinical features of these patients in a structured and uniform way, the information will become increasingly valuable. Fortunately, this is becoming easier due to advances in tools and software applications, and many professionals in the academic and commercial world have agreed to collaborate in order to substantially increase the genotype/phenotype collection and make these anonymised data publicly available to medical professionals in order to improve patient care worldwide”, Dr. de Leeuw concluded.

###

Abstract no: C04.5, Session: Sunday, June 24, 1:15 pm

[1] A DNA microarray (or gene chip) is a collection of microscopic DNA spots attached to a solid surface, used to measure the relative quantity of large numbers of genes simultaneously in multiple regions of a genome, thereby determining the DNA copy number.

[2] A SNP (pronounced ‘snip’), or single nucleotide polymorphism, is a relatively common DNA sequence variation, a change in a single nucleotide A, T, C or G in the genome, without clinical consequences. Each person has about 3 million SNPs in his or her genome, a unique genetic signature. In a SNP microarray, the probes (small pieces of DNA) bind only to the complementary piece of DNA being analysed if it is an exact match, making it possible to determine the genotype (SNP composition) of a DNA sample in addition to the DNA copy number.

---

[ | E-mail | Share ]

?

AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.

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Letters to the Editor for the weekly print issue of June 25, 2012: Saudi Arabia’s oppression of women, foreign workers, and the Shiite minority is shocking;?Don’t jump to faulty conclusions to explain an uptick in traffic accidents after a ban on texting while driving.

June 25, 2012

Saudi Arabia isn’t a model for reform

The June 4 Focus article “Reformers in the land of Mecca” notes progress in the fight for democratic reforms and the battle to improve human rights in Saudi Arabia. But the article fails to fully convey the level of oppression many in Saudi Arabia ? especially women and religious minorities ? continue to face daily.

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We must not forget: The degree of repression against women, foreign workers, and the Shiite minority in Saudi Arabia is shocking by American standards. (Yet the word “Shiite” does not even appear in the article!)

In my view, not fully acknowledging these problems amounts to disinformation or lying by omission.

Don Bedell

Makanda, Ill.

Faulty logic, unfair characterizations

The headline for Jim Sollisch’s June 4 commentary (“Why would anyone oppose a ban on texting?”) is thought provoking. But in my opinion, his attempts at cleverness fall flat.

I don’t buy into his characterization or humor when he calls those opposing a ban on texting while driving “The Slippery Slope Party,” claiming they argue that such a government restriction on personal liberty is the first step leading to a “totalitarian state.”

Similarly, his logic strikes me as faulty when he offers an explanation for the uptick in traffic accidents after texting bans had been enacted in certain states.

Mr. Sollisch claims that making texting illegal won’t prevent people from doing it; they’ll simply put cellphones in their laps, leading to increased accidents. Such an interpretation of statistics is weak (for one, a small sample size frequently leads to false results/conclusions).

Peter Bowman

Kittery, Maine

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This week on Double Feature Friday, there are courageous, redheaded warriors and their realistic counterparts, two fictional movies about two very real presidents, and two very different looks at the end of the world. Check out all your week’s pairings on Double Feature Friday! “Brave” & “Braveheart” In the pantheon of cinematic Scottish heroes, none [...]

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Hello, I am The Great Thundorz. I have made two roleplays so far, hoping for this to become the third. I am taking this chance to test out the interest check and to try out a sort of open world roleplay. I have decided to call the roleplay Destiny or Fate to represent one of the themes of the RP.

So, what will you as the player be able to do and how will it matter in the RP? The nice thing for you is, there are no set roles within it. You can start off as a good and honest person, a nasty and hate filled person, all up to you! D or F’s main storyline also gives you as the player a purpose to go off on an adventure, again, not mattering what your intentions are. Already your name will become big for doing this, but what you do on the journey is the most important thing for gaining reputation, or even losing reputation if you prefer to remain hidden.

I can’t seem to come up with anymore to say about it, but I do have one more thing to show if your still not interested. I will let the roleplay do the talking for me now, and show you a part of what will come, while also revealing some of the main storyline, and maybe even some about the world of D or F.

— Our time was over when the last war ended, and the king of our world was finally slain. He had plunged himself into the darkness, in his own attempt to save us, and corrupted the land. He had no wife or heir, and the people fought with one another over who should take the throne in his place. The Observer, our guardian and hero of the war, took the old king’s sword and broke it into six pieces. Each piece but one was reforged into a new but weaker sword, and the Observer hid them away around the land. When he appeared before us, he told us of a way to settle who would become the one to sit in the throne. ‘Whoever shall find the final piece and pulls it from it’s resting place shall become the ruler of the land. To find this piece, you must find it’s five brothers and they’re chosen wielders.’

Leaving behind only a map of where to start, the entire nation went in search for the five swords. For 50 years, different people have found the swords and found the final piece, but none have been able to free it. Now with the five swords lost again, people have given up, leaving the final piece and it’s location behind. The Observer has also vanished, leaving behind a supposed son who once a year takes a group of champions with him. Only time will tell now whether the land will further plunge into darkness or whether someone will be able to pull out the final piece. —

jane fonda morgan freeman jon huntsman bit coin huntsman w.e. episodes